Scientists at Harvard University and Massachusetts General Hospital have transplanted a small number of neurons in the brains of obese mice. In doing so they seek to restore brain function, particularly the repair of the leptin receptor that allows one to feel hunger and satiety. Without this leptin sensor, one does not get a sense of satiety, often leading to obesity. The problem encountered in the past with transplanting neurons is that the cells usually grow uncontrollably, causing cancer. In this case, the obese mice were able to lose weight and maintain a healthy weight as well as reverse past metabolic damage. This study promises the possibility of new repair therapy in the future.
http://www.sciencedaily.com/releases/2011/11/111125161023.htm
-Lindsey May
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